SS-31

SS-31 has demonstrated cardioprotective properties in preclinical studies and animal models. It has been shown to reduce mitochondrial dysfunction, decrease cell death, and improve cardiac function following ischemia-reperfusion injury, a condition characterized by temporary restriction and restoration of blood flow to the heart. SS-31’s ability to preserve mitochondrial function may help mitigate tissue damage and improve outcomes in cardiac conditions.

SS-31 has also shown neuroprotective effects in various models of neurodegenerative diseases and acute brain injury. It may help protect neurons from oxidative damage, reduce inflammation, and improve mitochondrial function in the brain. These neuroprotective properties suggest potential applications for SS-31 in the treatment of conditions such as Alzheimer’s disease, Parkinson’s disease, and stroke.

SS-31 has been reported to possess anti-inflammatory properties by modulating inflammatory signaling pathways. By reducing inflammation, SS-31 may help mitigate tissue damage and support tissue repair processes. This anti-inflammatory activity may contribute to SS-31’s therapeutic benefits in various disease conditions characterized by inflammation and tissue injury.

Some research suggests that SS-31 may have renoprotective effects, particularly in the context of acute kidney injury and chronic kidney disease. SS-31 has been shown to preserve mitochondrial function in kidney cells, reduce oxidative stress, and attenuate kidney injury in animal models. These findings suggest potential applications for SS-31 in protecting kidney function and preventing kidney damage.